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AP-1 - The Jun proteins: Oncogenes or tumor suppressors in disguise?
Eitan Shaulian
*
*
Corresponding author for this work
Department of Biochemistry and Molecular Biology
Research output
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Contribution to journal
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Review article
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peer-review
370
Scopus citations
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Biochemistry, Genetics and Molecular Biology
Tumor Suppressor Protein
100%
Cell Survival
100%
Transcription
100%
Cell Cycle
100%
Carcinogenesis
100%
Synapsin I
100%
Oncogene Protein
100%
Cell Migration
50%
Cell Proliferation
50%
Animal Model
50%
Wound Healing
50%
Programmed Cell Death
50%
Keyphrases
Tumor Suppressor
100%
Oncogene
100%
Activator protein-1 (AP-1)
100%
C-Jun
100%
JunB
66%
Neoplasia
33%
Cellular Survival
33%
Apoptosis
16%
Inflammation
16%
In Cancer
16%
Human Cancer
16%
Gene Coding
16%
Wound Healing
16%
Cell Proliferation
16%
Lymphoma
16%
Cyclin D
16%
Oncogenic Role
16%
Dogma
16%
P16INK4a
16%
Negative Regulation
16%
Negative Regulator
16%
P19Arf
16%
Oncogenic Pathways
16%
Cellular Migration
16%
Hodgkin
16%
Antagonistic Activity
16%
Positive Regulation
16%
CyclinA
16%
Cell Cycle Regulators
16%
Pharmacology, Toxicology and Pharmaceutical Science
Neoplasm
100%
Malignant Neoplasm
100%
Carcinogenesis
100%
Synapsin I
100%
Oncoprotein
100%
Tumor Suppressor Protein
100%
Inflammation
50%
Wound Healing
50%
Immunology and Microbiology
Cell Survival
100%
Cell Cycle
100%
Oncogene
100%
Cell Migration
50%
Wound Healing
50%
Cell Proliferation
50%
Programmed Cell Death
50%