TY - JOUR
T1 - Corrigendum to “Naphthoquinone-tyrptophan reduces neurotoxic Aβ*56 levels and improves cognition in Alzheimer's disease animal model” Neurobiol Dis. 46(3) (2012):663-72
AU - Attali, R. Scherzer
AU - Farfara, D.
AU - Cooper, I.
AU - Levin, A.
AU - Ben-Romano, T.
AU - Trudler, D.
AU - Vientrov, M.
AU - Shaltiel-Karyo, R.
AU - Shalev, D. E.
AU - Segev-Amzaleg, N.
AU - Gazit, E.
AU - Segal, D.
AU - Frenkel, D.
N1 - Publisher Copyright:
© 2025 Elsevier Inc. All rights are reserved, including those for text and data mining, AI training, and similar technologies.
PY - 2025
Y1 - 2025
N2 - The authors regret that we have become aware that while paper [1] was cited in this publication as a previous work, it was not explicitly specified that the control of Aβ only (without inhibitor) presented in Fig. 1C and D was the same one used in paper [1] in Fig. 1A and 1C as a control (without inhibotor). They reflect one set of experiments performed by the first author of the two publications that included two different inhibitors (naphthoquinone-tyrptophan in paper [1] and chloro-naphthoquinone-tyrptophan in the current publication [2]) and one control. The authors would like to apologise for any inconvenience caused. [1] Scherzer-Attali, R., Pellarin, R., Convertino, M., Frydman-Marom, A., Egoz-Matia, N., Peled, S., Levy-Sakin, M., Shalev, D.E., Caflisch, A., Gazit, E. and Segal, D., 2010. Complete phenotypic recovery of an Alzheimer's disease model by a quinone-tryptophan hybrid aggregation inhibitor. PloS One, 5(6), 11101.
AB - The authors regret that we have become aware that while paper [1] was cited in this publication as a previous work, it was not explicitly specified that the control of Aβ only (without inhibitor) presented in Fig. 1C and D was the same one used in paper [1] in Fig. 1A and 1C as a control (without inhibotor). They reflect one set of experiments performed by the first author of the two publications that included two different inhibitors (naphthoquinone-tyrptophan in paper [1] and chloro-naphthoquinone-tyrptophan in the current publication [2]) and one control. The authors would like to apologise for any inconvenience caused. [1] Scherzer-Attali, R., Pellarin, R., Convertino, M., Frydman-Marom, A., Egoz-Matia, N., Peled, S., Levy-Sakin, M., Shalev, D.E., Caflisch, A., Gazit, E. and Segal, D., 2010. Complete phenotypic recovery of an Alzheimer's disease model by a quinone-tryptophan hybrid aggregation inhibitor. PloS One, 5(6), 11101.
UR - https://www.scopus.com/pages/publications/105023847161
U2 - 10.1016/j.nbd.2025.107170
DO - 10.1016/j.nbd.2025.107170
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C2 - 41206330
AN - SCOPUS:105023847161
SN - 0969-9961
JO - Neurobiology of Disease
JF - Neurobiology of Disease
ER -