Corrigendum to “Naphthoquinone-tyrptophan reduces neurotoxic Aβ*56 levels and improves cognition in Alzheimer's disease animal model” Neurobiol Dis. 46(3) (2012):663-72

  • R. Scherzer Attali
  • , D. Farfara
  • , I. Cooper
  • , A. Levin
  • , T. Ben-Romano
  • , D. Trudler
  • , M. Vientrov
  • , R. Shaltiel-Karyo
  • , D. E. Shalev
  • , N. Segev-Amzaleg
  • , E. Gazit
  • , D. Segal
  • , D. Frenkel

Research output: Contribution to journalComment/debate

Abstract

The authors regret that we have become aware that while paper [1] was cited in this publication as a previous work, it was not explicitly specified that the control of Aβ only (without inhibitor) presented in Fig. 1C and D was the same one used in paper [1] in Fig. 1A and 1C as a control (without inhibotor). They reflect one set of experiments performed by the first author of the two publications that included two different inhibitors (naphthoquinone-tyrptophan in paper [1] and chloro-naphthoquinone-tyrptophan in the current publication [2]) and one control. The authors would like to apologise for any inconvenience caused. [1] Scherzer-Attali, R., Pellarin, R., Convertino, M., Frydman-Marom, A., Egoz-Matia, N., Peled, S., Levy-Sakin, M., Shalev, D.E., Caflisch, A., Gazit, E. and Segal, D., 2010. Complete phenotypic recovery of an Alzheimer's disease model by a quinone-tryptophan hybrid aggregation inhibitor. PloS One, 5(6), 11101.

Original languageEnglish
JournalNeurobiology of Disease
DOIs
StateAccepted/In press - 2025

Bibliographical note

Publisher Copyright:
© 2025 Elsevier Inc. All rights are reserved, including those for text and data mining, AI training, and similar technologies.

Fingerprint

Dive into the research topics of 'Corrigendum to “Naphthoquinone-tyrptophan reduces neurotoxic Aβ*56 levels and improves cognition in Alzheimer's disease animal model” Neurobiol Dis. 46(3) (2012):663-72'. Together they form a unique fingerprint.

Cite this