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Ectopic over expression of kiss1 may compensate for the loss of kiss2
Talya Etzion
, Nilli Zmora
, Yonatan Zohar
,
Berta Levavi-Sivan
, Matan Golan
, Yoav Gothilf
*
*
Corresponding author for this work
Department of Animal Sciences
Research output
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Contribution to journal
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Article
›
peer-review
11
Scopus citations
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Biochemistry, Genetics and Molecular Biology
Zebra Fish
100%
Messenger RNA
100%
Kisspeptin
100%
Wild Type
50%
Isoform
50%
CRISPR
50%
Genetic Manipulation
50%
Transcription Activator-Like Effector Nuclease
50%
In Situ Hybridization
50%
Neuropeptide
50%
Neuroscience
Hypothalamus
100%
Kisspeptin
100%
Messenger RNA
100%
In Situ Hybridization
50%
Habenula
50%
Neuropeptide
50%
CRISPR
50%
Transcription Activator-Like Effector Nuclease
50%
Nucleus
50%
Keyphrases
Kiss1
100%
Kiss2
100%
Ectopic Overexpression
100%
Hypothalamus
22%
Zebrafish
22%
Further Analysis
11%
Loss Function
11%
Expression Level
11%
Genetic Manipulation
11%
Clustered Regularly Interspaced Short Palindromic Repeats (CRISPR)
11%
Reproductive Capacity
11%
In Situ Hybridization
11%
Habenula
11%
Neuropeptides
11%
Hybrid Analysis
11%
Compensatory Mechanisms
11%
Hypothalamic-pituitary-gonadal Axis
11%
Kisspeptin
11%
Function Approach
11%
Knockout Lines
11%
Paralogous Genes
11%
TALEN
11%
Kisspeptin Gene
11%
Non-mammalian Vertebrates
11%
Entopeduncular nucleus
11%
Ventral Telencephalon
11%
Palindromic Repeat
11%
Sexual Fertility
11%
Pharmacology, Toxicology and Pharmaceutical Science
Zebra Fish
100%
Kisspeptin
100%
Messenger RNA
100%
Neuropeptide
50%
Transcription Activator Like Effector Nuclease
50%
Veterinary Science and Veterinary Medicine
Hypothalamus
100%
Kisspeptin
100%
Protocerebrum
50%
Habenula
50%
Neuropeptide
50%
In Situ Hybridization
50%