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Exploiting the therapeutic potential of leptin signaling in cachexia
Robert H. Mak
*
, Wai W. Cheung
,
Arieh Gertler
*
Corresponding author for this work
Institute of Biochemistry, Food Science and Nutrition
Research output
:
Contribution to journal
›
Review article
›
peer-review
6
Scopus citations
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Keyphrases
Therapeutic Potential
100%
Chronic Kidney Disease
100%
Cachexia
100%
Leptin Signaling
100%
Disease-associated
50%
Leptin
33%
Leptin Antagonist
33%
Congestive Heart Failure
16%
Therapeutic Strategies
16%
PEGylated
16%
Chronic Disease
16%
Chronic Obstructive Pulmonary Disease
16%
Antagonism
16%
Leptin Levels
16%
Leptin Receptor
16%
Melanocortin 4 Receptor (MC4R)
16%
Poor nutritional Status
16%
PEGylation
16%
Antagonist Treatment
16%
Cancer Cachexia
16%
Db Mice
16%
Chronic Kidney Disease Patient
16%
Pharmacology, Toxicology and Pharmaceutical Science
Chronic Kidney Failure
100%
Leptin
100%
Cachexia
100%
Syndrome
14%
Malignant Neoplasm
14%
Congestive Heart Failure
14%
Morbidity
14%
Leptin Receptor
14%
Anorexia
14%
Chronic Obstructive Lung Disease
14%
Melanocortin 4 Receptor
14%
PEGylation
14%
Db/Db Mouse
14%