HMGN1 modulates nucleosome occupancy and dnase i hypersensitivity at the CpG island promoters of embryonic stem cells

Tao Deng, Zhu Z. Iris, Shaofei Zhang, Fenfei Leng, Srujana Cherukuri, Loren Hansen, Leonardo Mariño-Ramírez, Eran Meshorer, David Landsman, Michael Bustina*

*Corresponding author for this work

Research output: Contribution to journalArticlepeer-review

34 Scopus citations

Abstract

Chromatin structure plays a key role in regulating gene expression and embryonic differentiation; however, the factors that determine the organization of chromatin around regulatory sites are not fully known. Here we show that HMGN1, a nucleosomebinding protein ubiquitously expressed in vertebrate cells, preferentially binds to CpG island-containing promoters and affects the organization of nucleosomes, DNase I hypersensitivity, and the transcriptional profile of mouse embryonic stem cells and neural progenitors. Loss of HMGN1 alters the organization of an unstable nucleosome at transcription start sites, reduces the number of DNase I-hypersensitive sites genome wide, and decreases the number of nestin-positive neural progenitors in the subventricular zone (SVZ) region of mouse brain. Thus, architectural chromatin-binding proteins affect the transcription profile and chromatin structure during embryonic stem cell differentiation

Original languageEnglish
Pages (from-to)3377-3389
Number of pages13
JournalMolecular and Cellular Biology
Volume33
Issue number16
DOIs
StatePublished - 2013

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