In computational models, action potential propagation in ultrathin axons is resilient despite considerable intracellular Na+ accumulation

Oron Kotler; Michael J. Gutnick; Ilya A. Fleidervish

doi:10.1073/pnas.2120782119

In computational models, action potential propagation in ultrathin axons is resilient despite considerable intracellular Na⁺ accumulation

Oron Kotler, Michael J. Gutnick^*, Ilya A. Fleidervish^*

^*Corresponding author for this work

The Koret School of Veterinary Medicine

Research output: Contribution to journal › Letter › peer-review

2 Scopus citations

Original language	English
Article number	e2120782119
Journal	Proceedings of the National Academy of Sciences of the United States of America
Volume	119
Issue number	12
DOIs	https://doi.org/10.1073/pnas.2120782119
State	Published - 22 Mar 2022

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10.1073/pnas.2120782119

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title = "In computational models, action potential propagation in ultrathin axons is resilient despite considerable intracellular Na+ accumulation",

author = "Oron Kotler and Gutnick, {Michael J.} and Fleidervish, {Ilya A.}",

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In computational models, action potential propagation in ultrathin axons is resilient despite considerable intracellular Na⁺ accumulation. / Kotler, Oron; Gutnick, Michael J.; Fleidervish, Ilya A.
In: Proceedings of the National Academy of Sciences of the United States of America, Vol. 119, No. 12, e2120782119, 22.03.2022.

Research output: Contribution to journal › Letter › peer-review

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In computational models, action potential propagation in ultrathin axons is resilient despite considerable intracellular Na+ accumulation

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In computational models, action potential propagation in ultrathin axons is resilient despite considerable intracellular Na⁺ accumulation