Inactivation of Snt2, a BAH/PHD-containing transcription factor, impairs pathogenicity and increases autophagosome abundance in Fusarium oxysporum

Keyphrases

• Neurospora Crassa 100%
• Transcription Factor 100%
• Pathogenicity 100%
• Autophagosome 100%
• Fusarium Oxysporum 100%
• Wilt Disease 66%
• Quantitative PCR 33%
• Overexpression 33%
• Sexual Development 33%
• Morphological Abnormalities 33%
• Gene Products 33%
• Polymerase Chain Reaction Analysis 33%
• Causal Agent 33%
• Disease Symptoms 33%
• Agrobacterium-mediated Transformation 33%
• Hybrid Analysis 33%
• Hyphal 33%
• Kinase Cascade 33%
• Schizosaccharomyces Pombe 33%
• Vegetative Growth 33%
• Muskmelon 33%
• Targeted Disruption 33%
• Fusarium Oxysporum F. Sp 33%
• Autophagy Pathway 33%
• Suppression Subtractive Hybridization 33%
• Soil-borne 33%
• Host Colonization 33%
• Septation 33%
• Biomass Accumulation 33%
• Asexual Fungi 33%
• Target of Rapamycin Kinase 33%
• Conidia Production 33%
• Perithecia 33%
• Wild-type Isolates 33%

Agricultural and Biological Sciences

• Pathogenicity 100%
• Fusarium oxysporum 100%
• Wilt Disease 100%
• Autophagosome 100%
• Transcription Factors 100%
• Plantae 50%
• Reverse Transcription Polymerase Chain Reaction 50%
• Plant-Based Food 50%
• Conidium 50%
• Autophagy 50%
• Agrobacterium tumefaciens 50%
• Neurospora crassa 50%
• Muskmelon 50%
• Vegetative Growth 50%
• Perithecia 50%
• Schizosaccharomyces Pombe 50%
• Gene Product 50%

Immunology and Microbiology

• Transcription Factors 100%
• Pathogenicity 100%
• Autophagosome 100%
• Fusarium oxysporum 100%
• Neurospora crassa 50%
• Morphology 50%
• Autophagy 50%
• Wild Type 50%
• Gene Product 50%
• Sirolimus 50%
• Quantitative Reverse Transcription Polymerase Chain Reaction 50%
• Schizosaccharomyces Pombe 50%
• Agrobacterium tumefaciens 50%
• Vegetative Growth 50%
• Perithecium 50%
• Plant-Based Food 50%
• Conidium 50%