Insulin Modulates NK Cell Activity in Liver Fibrosis MASH Patients via the STING Pathway

Keyphrases

• Insulin 100%
• Liver Fibrosis 100%
• Natural Killer Cell Activity 100%
• Stimulator of Interferon Genes (STING) 100%
• Metabolic Dysfunction-associated Steatohepatitis 100%
• Stimulator of Interferon Genes Pathway 100%
• Natural Killer Cells 80%
• Gene Expression 70%
• CGAMP 30%
• Cytotoxicity 20%
• Natural Killer Cell Cytotoxicity 20%
• Insulin Receptor 20%
• Advanced Fibrosis 20%
• Stimulator of Interferon Genes Agonist 20%
• Healthy Controls 10%
• Immune Cells 10%
• Flow Cytometry 10%
• Disease Severity 10%
• Interferon-α (IFN-α) 10%
• Protein Level 10%
• NKp46 10%
• Fibrosis 10%
• Functional Activity 10%
• Interferon Production 10%
• Therapeutic Approaches 10%
• Immune Surveillance 10%
• Insulin Signaling Pathway 10%
• IFN Production 10%
• Defense System 10%
• Activation Markers 10%
• Hepatic Stellate Cell Activation 10%
• Innate Immune Defense 10%
• Insulin Signaling 10%
• Siglec-7 10%
• Insulin Therapy 10%
• Apoptosis Assay 10%
• DNA Sensing 10%
• DMXAA 10%
• Agonist Treatment 10%
• Insulin Receptor Signaling 10%
• Fibrosis Stage 10%
• DNA Typing 10%
• CD107a 10%
• LAIR-1 10%
• Gene Signaling 10%
• Stimulator of Interferon Gene Protein 10%

Immunology and Microbiology

• Natural Killer Cell 100%
• Steatohepatitis 100%
• Stimulator of Interferon Genes 100%
• Insulin 100%
• Gene Expression 41%
• Cytotoxicity 23%
• Cell Function 17%
• Insulin Receptor 17%
• Agonist 11%
• Cell Activation 11%
• Insulin Signaling 11%
• Signal Transduction 5%
• Immunocompetent Cell 5%
• Interferon Production 5%
• Immunosurveillance 5%
• Sialic Acid Binding Immunoglobulin Like Lectin 5%
• Hepatic Stellate Cell 5%
• Programmed Cell Death 5%