Meta-analysis of genetic and environmental Parkinson's disease models reveals a common role of mitochondrial protection pathways

Keyphrases

• Meta-analysis 100%
• Metal Ions 100%
• Neuroinflammation 100%
• Parkinson's Disease 100%
• 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine 100%
• Parkinson's Disease Model 100%
• Mitochondrial Protection 100%
• Environmental Risk 50%
• Environmental Factors 50%
• Neurodegenerative Diseases 50%
• Protective Factors 50%
• Late-onset 50%
• Alpha-synuclein (α-syn) 50%
• Brain Regions 50%
• Alternative Splicing 50%
• Gene-environment Interaction 50%
• Genetic Factors 50%
• Functional Analysis 50%
• Chaperone 50%
• Heat Shock Protein 70 (HSP70) 50%
• Transcriptome 50%
• Overexpressing 50%
• Gene Ontology 50%
• Nuclear factor-κB 50%
• Acetylcholinesterase 50%
• Genetic Risk 50%
• Neurotoxin 50%
• Self-organizing Map 50%
• Early Onset 50%
• Gene Expression Changes 50%
• Multiple Treatments 50%
• Underlying Disease 50%
• Associated Genes 50%
• Parkinsonian 50%
• Mitochondrial Pathway 50%
• Shared Risk 50%
• Ontological Analysis 50%
• Map Classification 50%
• Functional Enrichment 50%
• Sporadic PD 50%

Biochemistry, Genetics and Molecular Biology

• Genetics 100%
• Gene Expression 50%
• Genetic Risk 50%
• Synuclein 50%
• Transcriptome 50%
• Alternative Splicing 50%
• Gene Ontology 50%
• Neurotoxin 50%
• Dopaminergic 50%
• Acetylcholinesterase 50%
• Gene-Environment Interaction 50%
• Immunity 50%

Neuroscience

• Parkinson's Disease 100%
• Meta-Analysis 100%
• Tetrahydropyridine 66%
• Neuro-Inflammation 66%
• Gene Expression 33%
• Synuclein 33%
• Acetylcholinesterase 33%
• Transcriptome 33%
• Dopaminergic 33%
• Alternative Splicing 33%
• Chaperone 33%
• Neurotoxin 33%