Pancreatic Lkb1 deletion leads to acinar polarity defects and cystic neoplasms

Medicine and Dentistry

• Cell Polarity 100%
• Acinar Cell 100%
• Cystic Neoplasm 100%
• Neoplasm 66%
• Hydroxymethylglutaryl Coenzyme A Reductase Kinase 66%
• Peutz Jeghers Syndrome 66%
• Homeostasis 33%
• Disease Predisposition 33%
• Cell Viability 33%
• Epithelial Cell 33%
• Pancreatic Insufficiency 33%
• Kinase 33%
• Pancreas Cancer 33%
• Cell Function 33%
• Tight Junction 33%
• Endocrine Pancreas 33%
• Germ Line 33%
• Loss of Function Mutation 33%
• Metaplasia 33%
• Cystadenoma 33%
• Cancer Inhibition 33%
• Cell Degeneration 33%
• Gastrointestinal Tumor 33%
• Germ Cell 33%

Keyphrases

• Liver Kinase B1 (LKB1) 100%
• Cystic Neoplasm 100%
• Cell Polarity 25%
• Pancreas 25%
• Acinar Cells 25%
• AMP-activated Protein Kinase 25%
• Peutz-Jeghers Syndrome 25%
• High Risk 12%
• Neoplasia 12%
• Tumor Subtype 12%
• Tumor Suppression 12%
• Germ Cells 12%
• Tight Junction 12%
• Epithelium 12%
• Energy Homeostasis 12%
• Islet 12%
• Cell Function 12%
• Physiological Function 12%
• Acinar-to-ductal Metaplasia 12%
• Pancreatic Acinar Cells 12%
• Cell Viability 12%
• Genetic Studies 12%
• Pancreatic Insufficiency 12%
• Pancreatic Cancer 12%
• In Vivo Evidence 12%
• Gain-of-function mutation 12%
• Cell Tumor 12%
• At Birth 12%
• Vascular Development 12%
• Cytoskeletal Organization 12%
• Cell Degeneration 12%
• Pancreatic Serous Cystadenoma 12%
• Epithelial Polarity 12%
• Gastrointestinal Neoplasms 12%
• Glucose Control 12%
• Developmental Function 12%