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RIC-3 expression and splicing regulate nAChR functional expression
Yael Ben-David
, Tehila Mizrachi
, Sarah Kagan
, Tamar Krisher
, Emiliano Cohen
, Talma Brenner
,
Millet Treinin
*
*
Corresponding author for this work
Department of Medical Neurobiology
Research output
:
Contribution to journal
›
Article
›
peer-review
22
Scopus citations
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Keyphrases
RIC-3
100%
Functional Expression
100%
Nicotinic Acetylcholine Receptor (nAChR)
100%
Immune Cells
37%
Quantitative PCR
25%
Expression Level
25%
Inflammation
12%
Central Nervous System
12%
Gene Regulation
12%
Alternative Splicing
12%
Chaperone
12%
Endoplasmic Reticulum
12%
Inflammatory Pathways
12%
Large Families
12%
Drosophila Melanogaster
12%
Xenopus Oocytes
12%
In Situ Hybridization
12%
Through Effect
12%
Acetylcholine
12%
Cellular Factors
12%
Diverse Families
12%
Diverse Roles
12%
Gated Ion Channels
12%
Electrophysiological Analysis
12%
Cholinergic Anti-inflammatory Pathway
12%
Neuroinflammatory Diseases
12%
Neuroscience
Nicotinic Acetylcholine Receptor
100%
Anti-Inflammatory
14%
Cholinergic
14%
In Situ Hybridization
14%
Ion Channel
14%
Central Nervous System
14%
Real-Time Polymerase Chain Reaction
14%
Acetylcholine
14%
Drosophila Melanogaster
14%
Chaperone
14%
Endoplasmic Reticulum
14%