Severe thalassaemia intermedia caused by interaction of homozygosity for α-globin gene triplication with heterozygosity for β° thalassaemia

V. Oron, D. Filon, A. Oppenheim, D. Rund*

*Corresponding author for this work

Research output: Contribution to journalArticlepeer-review

39 Scopus citations

Abstract

A 3-year-old child was evaluated for β-thalassaemia intermedia. Molecular characterization including β-globin gene sequence analysis revealed heterozygosity for a single β-thalassaemia mutation, IVSI nt1 (G→A). In addition the patient was found to be homozygous for α-globin gene triplication (ααα(anti 3.7)/ααα(anti 3.7)). The propositus has a significantly more severe phenotype than has been previously reported with this combination of genetic defects. In contrast, four individuals heterozygous for both triplicated α and for β thalassaemia had a phenotype of thalassaemia minor, and a fifth had very mild thalassaemia intermedia.

Original languageEnglish
Pages (from-to)377-379
Number of pages3
JournalBritish Journal of Haematology
Volume86
Issue number2
DOIs
StatePublished - 1994
Externally publishedYes

Keywords

  • homozygosity
  • triplicated α-globin genes
  • β thalassaemia

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