Specific inhibition of NADPH oxidase 2 modifies chronic epilepsy

Keyphrases

• Specific Inhibition 100%
• Chronic Epilepsy 100%
• NADPH Oxidase 2 (NOX2) 100%
• NADPH Oxidase 80%
• Gp91ds-tat 80%
• Epilepsy 60%
• Epileptiform Activity 60%
• Seizure 40%
• Status Epilepticus 40%
• Brain Insult 40%
• Reactive Oxygen Species Generation 40%
• Neuronal Damage 20%
• Ca2+ 20%
• Oxidative Stress 20%
• Hippocampus 20%
• Acid Activation 20%
• Reactive Oxygen Species 20%
• Mitochondrial Depolarization 20%
• Oscillation 20%
• Neuronal Development 20%
• Cortex 20%
• Epileptic Rats 20%
• Neuronal Death 20%
• Intracerebroventricular Administration 20%
• Activity-based Model 20%
• Competitive Inhibitor 20%
• Seizure Frequency 20%
• Temporal Lobe Epilepsy Model 20%
• Kainic Acid 20%
• Oxidase Enzymes 20%
• In Vitro Seizure Models 20%

Neuroscience

• Reduced Nicotinamide Adenine Dinucleotide Phosphate Oxidase 2 100%
• NADPH Oxidase 100%
• Epilepsy 100%
• Reactive Oxygen Species 75%
• In Vitro 50%
• Status Epilepticus 50%
• Cerebrovascular Accident 50%
• Hippocampus 25%
• Oxidative Stress 25%
• Temporal Lobe Epilepsy 25%
• Neuronal Cell Death 25%
• Kainic Acid 25%
• Enzymes 25%
• Competitive Inhibition 25%
• Peptide 25%
• Intracerebroventricular Drug Administration 25%

Pharmacology, Toxicology and Pharmaceutical Science

• Reduced Nicotinamide Adenine Dinucleotide Phosphate Oxidase 100%
• Reduced Nicotinamide Adenine Dinucleotide Phosphate Oxidase 2 100%
• Reactive Oxygen Metabolite 75%
• Epilepsy 75%
• Epileptic State 50%
• Cerebrovascular Accident 50%
• Rat 50%
• Calcium Ion 25%
• Temporal Lobe Epilepsy 25%
• Kainic Acid 25%
• Enzymes 25%
• Competitive Inhibitor 25%
• Peptide 25%
• Intracerebroventricular 25%