Sympathetic component of baroreflex control of heart rate is impaired in hypertension-prone (SBH) sabra rats

Donna Schorer-Apelbaum, Marta Weinstock*, Drori Ben-Lshay

*Corresponding author for this work

Research output: Contribution to journalArticlepeer-review

8 Scopus citations

Abstract

Baroreflex control of heart rate in response to phenylephrine was studied in conscious Sabra hypertension-prone (SBH) rats, at a prehypertensive stage, and hypertension-resistant (SBN) rats. Baroreflex sensitivity as determined from the slope of the relationship of mean arterial blood pressure and heart period was significantly lower in SBH rats (0.58 ± 0.06 versus 1.71 ± 0.11 ms/mmHg in SBN rats, P < 0.01) before the development of hypertension. Sympathetic nerve blockade with guanethidine (15 mg/kg) significantly reduced the slope of the mean arterial blood pressure-heart period relationship in SBN rats to 0.45 ± 0.05 ms/mmHg (P < 0.01) and increased the pressor response to phenylephrine, without having any effect on these parameters in SBH rats. Atropine methyl nitrate (1 mg/kg) abolished reflex vagal bradycardia in response to phenylephrine in both groups of rats. This suggests that SBH rats are unable to withdraw the sympathetic cardiac component of the baroreflex in response to a pressor stimulus and appear to rely only on increased vagal activity to effect bradycardia.

Original languageEnglish
Pages (from-to)257-260
Number of pages4
JournalJournal of Hypertension
Volume2
Issue number3
DOIs
StatePublished - Jun 1984

Keywords

  • Baroreflex sensitivity
  • Phenylephrine
  • Sabra hypertension-prone rats (SBH)
  • Sabra hypertension-resistant rats (SBN)
  • Sympathetic control
  • Vagai control

Fingerprint

Dive into the research topics of 'Sympathetic component of baroreflex control of heart rate is impaired in hypertension-prone (SBH) sabra rats'. Together they form a unique fingerprint.

Cite this