TRP channels in vision

Ben Katz; Richard Payne; Baruch Minke

doi:10.4324/9781315152837

TRP channels in vision

Ben Katz, Richard Payne, Baruch Minke

Institute for Medical Research Israel-Canada (IMRIC)

Research output: Chapter in Book/Report/Conference proceeding › Chapter › peer-review

17 Scopus citations

Abstract

The transient receptor potential (TRP) eld began (for reviews see Minke, 2010; Montell, 2011; Hardie, 2011) with the analysis of a spontaneously formed Drosophila mutant showing transient, rather than sustained, responses to prolonged intense illumination in electroretinogram (ERG) measurements, rendering the ies effectively blind (Cosens and Manning, 1969). Cosens and Manning (1969) isolated this mutant, designated this strain the “A-type” mutant, and attributed its phenotype to a failure of photopigment regeneration (Cosens, 1971). The isolation of this mutant, though potentially interesting, raised a number of concerns at the time. One main concern was that the results.

Original language	English
Title of host publication	Neurobiology of TRP Channels
Publisher	CRC Press
Pages	27-63
Number of pages	37
ISBN (Electronic)	9781498755269
ISBN (Print)	9781498755245
DOIs	https://doi.org/10.4324/9781315152837
State	Published - 1 Jan 2017

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© 2018 by Taylor & Francis Group, LLC.

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TRP channels in vision

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